Currently, CNV is classified according to its level of origin. In type 1 neovascularization, new vessels originating from the choriocapillaris grow through a defect in Bruch membrane into the sub–RPE space (Fig 4-6). Fluid leakage and bleeding can produce a vascularized serous or fibrovascular PED. These fibrovascular PEDs typically have an irregular surface contour. In type 2 neovascularization, the CNV originates between the RPE and the neurosensory retina. On examination, it typically appears as a lacy or gray-green lesion; in AMD, this finding is less common than type 1 neovascularization. In type 3 neovascularization, the neovascularization develops from the deep capillary plexus of the retina and grows downward toward the RPE. Because of their intraretinal origin, these lesions were originally termed retinal angiomatous proliferations (RAPs). On examination, they often appear as a small area of red discoloration, associated with retinal exudate or a bleb of subretinal fluid.