Your last comment does not at all follow from the preceding ones. Just because something causes massive genetic changes, doesn’t make it a viable “speciation mechanism.” As the Wiki article Peter referred to indicates, chromothripsis involves the sudden rearrangement of tens to thousands of chromosome fragments, and thus is associated with cancer and congenital diseases. So on what basis do you claim it an “ancient modeling” mechanism, and how is it helpful to your model, especially since you reject the adaptive role of natural selection? Without NS, even on the extremely remote chance chromothripsis will produce any kind of useful result or “hopeful monster” it’s a dead end. Moreover, the more massive any sudden change, the less likely it is going to be helpful or “hopeful”, even with NS. Often you misuse the term “speciation mechanism” to describe some generally random, massive, and/or destructuve process or event, without any means to make it useful or adaptive. In short, you don’t have an speciation mechanism, your have generally disruptive and destructive phenomena lacking a speciation mechanism.