Angiographic vasospasm has a typical temporal course, with onset at 3 to 5 days after the haemorrhage, maximal narrowing at 5 to 14 days and gradual resolution over 2 to 4 weeks[19],[24] In about 50% of the cases, vasospasm manifests by the occurrence of a delayed ischaemic neurological deficit (DIND), which may resolve or progress to infarction.[25] Cerebral vasospasm remains the greatest treatable cause of mortality and morbidity in patients who survive the ictus of SAH. In most contemporary series, up to 15% of such patients suffer stroke or death from vasospasm despite maximal therapy.[19],[24],[26],[27] In the present series 19 (70%) out of 27 patients with angiographic vasospasm manifested DIND during course of their illness. This high incidence of deterioration could probably be because of strict criteria applied for labelling a patient as having DIND. However, the 11 % mortality in patients with angiographic vasospasm compares favourably with other series. The international cooperative study on the timing of aneurysm surgery[19] reported that permanent morbidity and mortality resulting from vasospasm declined with increasing intervals to planned surgery, but a number of recent studies have not reported a worsening of vasospam after early surgery.[9],[28],[29] We also feel that angiographic vasospasm, or increased velocities on transcranial doppler did not significantly influence the outcome unless accompanied by poor clinical grade (grade IV) at the time of surgery.